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Most used

21
MELD-NaAssesses the severity of chronic liver diseaseChild-Pugh ScoreAssesses the prognosis of chronic liver disease, mainly cirrhosisFIB-4 IndexLiver fibrosis scoring indexAPRIAST to platelet ratio — liver fibrosisMaddrey's DFAlcoholic hepatitis severityGlasgow-BlatchfordUpper GI bleed risk stratificationGAHSGlasgow alcoholic hepatitis scoreMontreal IBDIBD classification — CD & UCMayo ScoreUlcerative colitis activityBISAP ScoreBedside index for severity of pancreatitisCLIF-SOFAOrgan failure scoring in cirrhosisAlcohol ContentStandard drinks & alcohol grams calculatorAARC-ACLFAcute-on-chronic liver failure gradePELD / CR ScorePediatric end-stage liver diseaseHarvey-BradshawCrohn's disease activity indexCTSICT severity index — pancreatitisRockall ScoreGI bleed rebleeding & mortality riskCAGEAlcohol use disorder screening (4 questions)MELD 3.0Updated MELD — sex-inclusive formulaAUDIT ScoreAlcohol use disorders identification testVOCAL-Penn ScorePost-operative mortality risk in cirrhosis surgery

Liver & Cirrhosis

17
ALBI GradeAlbumin-bilirubin liver function grade in HCCUKELD ScoreUK model for end-stage liver diseaseMELD-XIMELD excluding INR — for anticoagulated patientsWest Haven CriteriaHepatic encephalopathy gradingMilan CriteriaLiver transplant eligibility in hepatocellular carcinomaLI-RADS v2018 (CT/MRI)Liver observation category from size, APHE and major featuresBCLC StagingHepatocellular carcinoma stage and treatment allocationSimplified AIH CriteriaSimplified criteria for autoimmune hepatitisRevised Original AIH ScoreIAIHG 1999 comprehensive autoimmune hepatitis scoreSAAGSerum-ascites albumin gradient — cause of ascitesR FactorHepatocellular vs cholestatic pattern in liver injuryCLIF-C ACLFMortality prediction in acute-on-chronic liver failureKing's College CriteriaTransplant criteria in acute liver failureGALAD ScoreHCC detection from gender, age, AFP-L3, AFP and DCPMetroticket 2.0AFP-adjusted up-to-seven for HCC transplant eligibilityRUCAMCausality in drug- and herb-induced liver injuryBaveno VII CriteriacACLD, CSPH and sparing screening endoscopy

Fibrosis & MASLD

8
NAFLD Fibrosis ScoreAdvanced fibrosis probability in MASLD/NAFLDBARD ScoreBMI, AST/ALT ratio, diabetes — MASLD fibrosisFatty Liver IndexPredicts hepatic steatosis from routine labsFibrotic NASH Index (FNI)At-risk NASH probability from AST, HbA1c and HDLNAFLD Activity Score (NAS)Histologic activity grade — steatosis, inflammation, ballooningMEFIB IndexMRE + FIB-4 rule for significant fibrosis (≥F2) in MASLDFAST ScoreFibroScan-AST — at-risk NASH from LSM, CAP and ASTSAFE ScoreSteatosis-Associated Fibrosis Estimator for MASLD in primary care

Pancreas & Biliary

8
Ranson's CriteriaAcute pancreatitis severity at 48 hoursGlasgow-Imrie CriteriaAcute pancreatitis severity — the PANCREAS criteriaHAPSHarmless acute pancreatitis scoreTokyo Guidelines — CholangitisTG18 diagnosis and severity grade for acute cholangitisTokyo Guidelines — CholecystitisTG18 diagnosis and severity grade for acute cholecystitisBiliary Pain (Rome IV)Rome IV — defining biliary-type pain before interventionFunctional Pancreatic SODRome IV — pancreatic sphincter of Oddi disorderRevised Atlanta ClassificationAcute pancreatitis severity — mild, moderately severe, severe

IBD

9
Truelove & Witts CriteriaAcute severe ulcerative colitis — admission decisionUCEISUlcerative colitis endoscopic index of severitySCCAISimple clinical colitis activity index — symptoms onlyCDAICrohn's disease activity index — the trial standardSES-CDEndoscopic severity in Crohn's diseasePUCAIPaediatric ulcerative colitis activity indexTravis (Oxford) CriteriaDay 3 colectomy risk in acute severe ulcerative colitisHo IndexDay 3 steroid failure risk in acute severe ulcerative colitisRutgeerts ScorePostoperative Crohn's recurrence at ileocolonoscopy

GI Bleeding

7
EVendo ScorePredicts oesophageal varices needing treatmentForrest ClassificationPeptic ulcer bleeding — rebleeding risk at endoscopyAIMS65 ScoreUpper GI bleed mortality — five bedside criteriaOakland ScoreSafe-discharge risk for acute lower GI bleedingABC ScoreAge, blood tests, comorbidities — GI bleed mortalitySarin ClassificationEndoscopic classification of gastric varicesEGUS (Gastric Ulcer)Malignancy risk in a gastric ulcer, and who needs repeat endoscopy

Alcohol

2
ABIC ScoreAge, bilirubin, INR, creatinine — alcoholic hepatitisLille ModelSteroid response at day 7 in alcoholic hepatitis

Upper GI

4
Chicago Classification v4.0Oesophageal motility pattern from high-resolution manometryLA Classification (Oesophagitis)Los Angeles grade A–D for erosive oesophagitisPrague C & M CriteriaCircumferential and maximal extent of Barrett's oesophagusEREFS (Eosinophilic Oesophagitis)Endoscopic reference score — oedema, rings, exudates, furrows, stricture

Colorectal

3
Boston Bowel Prep ScaleColonoscopy preparation adequacy by segmentStool Osmotic GapOsmotic vs secretory diarrhoea from stool electrolytesATLAS Score (C. difficile)Predicted response to therapy in Clostridioides difficile infection

Functional GI

37
Bristol Stool ScaleStool form types 1–7 and colonic transitRome IV Criteria for IBSIrritable bowel syndrome diagnosis and subtypeFunctional ConstipationRome IV — two of six items, IBS excludedOpioid-Induced ConstipationRome IV — constipation tied to opioid therapyFunctional DiarrhoeaRome IV — loose stools without predominant painFunctional Bloating / DistensionRome IV — bloating without other bowel disorder criteriaUnspecified Functional Bowel DisorderRome IV — bowel symptoms fitting no other categoryCentrally Mediated Abdominal Pain (CAPS)Rome IV — continuous pain unrelated to gut eventsNarcotic Bowel SyndromeRome IV — opioid-induced hyperalgesia of the gutFaecal Incontinence (Rome IV)Rome IV — the criteria, and why nobody is askedFunctional Anorectal PainLevator ani, unspecified pain and proctalgia fugaxFunctional Defecation DisordersRome IV — dyssynergia and inadequate propulsionInfant RegurgitationRome IV — the happy spitter, and the alarm features that rule it outInfant ColicRome IV — recurrent unexplained crying in a well infant under 5 monthsInfant DyscheziaRome IV — straining before a soft stool, and why not to intervenePaediatric Functional ConstipationRome IV — two of six over one month, with overflow soiling as a criterionToddler's DiarrhoeaRome IV functional diarrhoea of childhood — painless, thriving childPaediatric Cyclic Vomiting SyndromeRome IV — both age bands, with different criteria for eachPaediatric Rumination SyndromeRome IV — infant and child/adolescent criteriaFunctional Nausea & Vomiting (Children)Rome IV — two separate disorders that can be met togetherAerophagiaRome IV — distension that increases through the dayPaediatric Functional DyspepsiaRome IV — four times a month, with PDS and EPS subtypingPaediatric Irritable Bowel SyndromeRome IV — plus the constipation clause clinicians missAbdominal MigraineRome IV — stereotypical incapacitating episodes weeks apartFunctional Abdominal Pain — NOSRome IV — the residual category, reached after the other threeNonretentive Faecal IncontinenceRome IV — soiling without retention, where laxatives make it worseFunctional DyspepsiaRome IV — with PDS and EPS subtypingRumination SyndromeRome IV — effortless regurgitation without retchingCyclic Vomiting SyndromeRome IV — stereotypical episodic vomitingCannabinoid HyperemesisRome IV — CVS pattern relieved by cannabis cessationChronic Nausea & VomitingRome IV — chronic nausea and vomiting syndromeBelching DisordersRome IV — supragastric vs gastric belchingFunctional HeartburnRome IV — heartburn with normal acid exposureReflux HypersensitivityRome IV — normal acid exposure, positive symptom associationFunctional Chest PainRome IV — non-cardiac, non-reflux chest painGlobusRome IV — painless lump-in-throat sensationFunctional DysphagiaRome IV — dysphagia with normal endoscopy and manometry
  1. Calculators
  2. /
  3. BCLC Staging
Liver & Cirrhosis

BCLC Staging

Hepatocellular carcinoma stage and treatment allocation

Performance status and liver function

Assessed first — either can override tumour burden entirely.

PS 1–2 places a patient in stage C even with a small tumour; PS 3–4 places them in stage D.

End-stage liver function places a patient in stage D whatever the tumour burden.

Vascular invasion and spread

Either finding places a patient in stage C.

Tumour burden

Only determines the stage when performance status is 0 and liver function is preserved.

A single nodule is stage 0 or A by size, however large. Stage B is more than 3 nodules, or 2–3 nodules with at least one above 3 cm.

For a single nodule the boundary is 2 cm (stage 0 versus A); for 2–3 nodules it is 3 cm (stage A versus B).

Assessed top-down: performance status and liver function are checked first, because either one on its own can place a patient in stage C or D regardless of how small the tumour is. Tumour burden only decides the stage once those are preserved.

When to use
Use it at diagnosis of hepatocellular carcinoma, and again at every reassessment, to link prognosis to a treatment recommendation. BCLC is unusual among cancer staging systems in that it does not stage the tumour alone — it combines tumour burden, liver function and the patient's physical condition, because all three constrain what treatment is possible in a cancer that almost always arises in a diseased liver. It presupposes a confirmed diagnosis of hepatocellular carcinoma, whether by non-invasive imaging criteria or histology, and does not apply to intrahepatic cholangiocarcinoma or to metastatic disease in the liver.
Why use it
Because in hepatocellular carcinoma the tumour is only one of three things that determine what can be done. A 2 cm lesion is curable in a patient with preserved liver function and untreatable in one with decompensated cirrhosis, and no purely anatomical staging system captures that. TNM stages the tumour and is a poor guide to therapy here; BCLC was built from the start to map each stage onto a treatment recommendation and a survival expectation, which is why it is the framework used by both EASL and AASLD. The 2026 update reinforces that design by adding stereotactic body radiotherapy and transarterial radioembolisation as potentially curative options at the early stages and consolidating combination immunotherapy as preferred first line in advanced disease.
Formula, evidence and interpretation

About the Barcelona Clinic Liver Cancer (BCLC) Staging System

Work top-down, because performance status and liver function can settle the stage before tumour burden is even considered. Performance status 3–4 or end-stage liver function is stage D; portal invasion, extrahepatic spread or performance status 1–2 is stage C. Only then does burden decide: a single nodule of 2 cm or less is stage 0, a single larger nodule or 2–3 nodules all 3 cm or less is stage A, and multifocal disease — more than 3 nodules, or 2–3 nodules with at least one above 3 cm — is stage B. That last definition is the clarification the 2026 update added. A single nodule stays stage A however large it is, which is the rule most often got wrong.

On this page

  • Formula
  • Interpreting the result
  • Inputs
  • What it returns
  • How it is calculated
  • Facts & figures
  • Evidence
  • How it compares
  • Pearls & pitfalls
  • Critical actions
  • Why it exists
  • About the creator
  • Limitations
  • If you are the patient
  • FAQ
  • Related calculators
  • References

Formula

Assessed in this order: 1. PS 3–4 OR end-stage liver function → Stage D 2. Portal invasion / extrahepatic spread OR PS 1–2 → Stage C 3. Otherwise (PS 0, preserved function), by burden: single nodule ≤ 2 cm → Stage 0 single nodule > 2 cm, or 2–3 nodules all ≤ 3 cm → Stage A > 3 nodules, or 2–3 nodules with one > 3 cm → Stage B
Performance status
ECOG 0–4. Checked first — it can override any tumour burden.
Liver function
Preserved (Child-Pugh A/B) versus end-stage (Child-Pugh C, not transplantable).
Multifocal (stage B)
More than 3 nodules, or 2–3 nodules with at least one above 3 cm. This wording is the clarification introduced in the 2026 update.
  • The order matters. A patient with a 1 cm nodule and Child-Pugh C cirrhosis is stage D, not stage 0 — liver function and performance status are assessed before burden, not alongside it.
  • A SINGLE nodule is stage 0 or A by size alone, however large. An 8 cm solitary tumour with PS 0 and preserved function is stage A, not stage B.
  • The two size boundaries apply to different situations: 2 cm separates 0 from A in a solitary nodule, and 3 cm separates A from B when there are 2 or 3.
  • The 2026 update preserves the 2022 stage definitions. What changed is the explicit definition of multifocal disease in stage B, plus treatment allocation within stages.
  • Stage D driven by liver function rather than by tumour burden or performance status is the one situation where transplantation may still offer a route out.

Interpreting the result

Read the stage together with what produced it. Stages 0 and A are curative-intent territory, where resection, ablation and transplantation should be considered together at a multidisciplinary meeting rather than sequentially, and where portal pressure and bilirubin often matter more than the tumour in choosing between them. Stage B is conventionally treated with transarterial chemoembolisation, though a proportion of patients fall within transplant criteria or can be downstaged into them. Stage C means systemic therapy, with combination immunotherapy consolidated as preferred first line in the 2026 update. Stage D means best supportive care — with one important exception: where the stage is driven by liver function rather than by tumour burden or performance status, transplantation may still be feasible, and that is the single most important question to ask before accepting a terminal designation. Across all stages, the 2026 update introduces the CUSE framework, which asks teams to weigh complexity, uncertainty, subjectivity and emotional factors explicitly rather than treating stage allocation as automatic.

ScoreBandWhat it meansAction
Stage 0Very earlySingle nodule ≤ 2 cm, PS 0, preserved liver function. Outcomes approach those of the general population with optimal treatmentAblation, or resection where anatomy favours it
Stage AEarlySingle nodule > 2 cm, or 2–3 nodules all ≤ 3 cm, PS 0, preserved function. Curative treatment is achievableResection, ablation or transplantation by liver function, portal pressure and anatomy; SBRT and TARE added as potentially curative in the 2026 update
Stage BIntermediateMultifocal disease — more than 3 nodules, or 2–3 with one > 3 cm — with PS 0 and preserved functionTransarterial chemoembolisation; assess against transplant criteria and consider downstaging
Stage CAdvancedPortal invasion and/or extrahepatic spread, or PS 1–2, with preserved liver functionSystemic therapy; combination immunotherapy consolidated as preferred first line in the 2026 update
Stage DTerminalAny tumour burden with PS 3–4 or end-stage liver functionBest supportive care — unless the stage is driven by liver function alone, in which case assess transplant candidacy

What the BCLC Staging needs (5 inputs)

ECOG performance status
0 to 4. PS 1–2 places a patient in stage C even with a tiny tumour; PS 3–4 places them in stage D. This is assessed before tumour burden, not after.
Liver function
Preserved (Child-Pugh A or B) or end-stage (Child-Pugh C and not transplantable). End-stage function is stage D whatever the tumour looks like.
Portal invasion and/or extrahepatic spread
Either finding is stage C. Confirm that portal involvement is tumour thrombus rather than bland thrombus — they are distinguishable on contrast imaging and carry different implications.
Number of nodules
Single, 2–3, or more than 3. A single nodule is stage 0 or A by size however large; more than 3 nodules is stage B regardless of size.
Largest nodule diameter
The boundary depends on the count: 2 cm separates stage 0 from stage A in a solitary nodule, and 3 cm separates stage A from stage B when there are 2 or 3.

What it returns

BCLC stage
0 (very early), A (early), B (intermediate), C (advanced) or D (terminal).
What determined the stage
Named explicitly, because a stage C driven by performance status alone is a different clinical conversation from one driven by portal invasion.
Treatment framing
The management approach associated with each stage in the 2026 update, as a starting point for multidisciplinary discussion rather than a substitute for it.

How it is calculated

BCLC treats hepatocellular carcinoma as a problem with three simultaneous constraints rather than one. Because the tumour almost always arises in a cirrhotic liver, the amount of functioning parenchyma limits what can be resected or ablated, and the patient's physical condition limits what they can tolerate. The system therefore evaluates performance status and liver function first, on the reasoning that either can make a technically treatable tumour untreatable in practice. Only when both are preserved does tumour burden decide the stage, and the burden thresholds are drawn where the evidence for particular treatments changes: 2 cm because ablation approaches resection in efficacy below it, and the Milan-adjacent limits because they mark where transplantation outcomes hold up. Each stage then carries a treatment recommendation and a survival expectation, which is the feature that distinguishes BCLC from purely anatomical staging systems and the reason both EASL and AASLD build their guidance around it.

Facts & figures

Stage definitions at a glance
StageTumour burdenPerformance statusLiver function
0 — very earlySingle nodule ≤ 2 cm0Preserved
A — earlySingle > 2 cm, or 2–3 nodules all ≤ 3 cm0Preserved
B — intermediate> 3 nodules, or 2–3 with one > 3 cm0Preserved
C — advancedPortal invasion and/or extrahepatic spread0–2Preserved
D — terminalAny3–4Any, or end-stage

Stages C and D are reached by performance status or liver function alone, without reference to burden — which is why those are assessed first.

What the 2026 update changed
AreaChange
Stage definitionsUnchanged from 2022
Stage B'Multifocal' explicitly defined as > 3 nodules, or ≤ 3 nodules with at least one > 3 cm
Stages 0 / AStereotactic body radiotherapy and transarterial radioembolisation introduced as potentially curative options alongside resection and ablation
Stage BEvidence held insufficient for routine locoregional plus systemic combination therapy
Stage CCombination immunotherapy further consolidated as preferred first line
FrameworkCUSE introduced — complexity, uncertainty, subjectivity and emotion weighed explicitly in multidisciplinary decisions

Because the stage definitions themselves are unchanged, a stage assigned under the 2022 update remains valid; what has moved is the treatment allocated within each stage.

Evidence

BCLC 2026 update — Reig, Forner et al.

2026

The current revision of the BCLC strategy, updating prognosis prediction and treatment recommendations while preserving the stage definitions established in the 2022 update.

Clarifies the definition of multifocal disease in stage B, introduces stereotactic body radiotherapy and transarterial radioembolisation as potentially curative options at stages 0 and A, consolidates combination immunotherapy as preferred first line at stage C, and introduces the CUSE framework for multidisciplinary decision-making.

BCLC 2022 update — the stage definitions in current use

2022

The 2022 revision, which established the stage definitions carried forward unchanged into 2026 and introduced the sub-stratification of intermediate-stage disease.

Refined stage B into groups by tumour burden and liver function, and set out the treatment-stage migration concept that allows patients to receive a treatment allocated to a different stage where the recommended option is unsuitable.

EASL clinical practice guidelines

2018

EASL clinical practice guidelines on the management of hepatocellular carcinoma, which adopt BCLC as the staging and treatment-allocation framework.

Establishes BCLC as the reference system for linking stage to treatment in European practice.

AASLD practice guidance

2018

AASLD guidance on the diagnosis, staging and management of hepatocellular carcinoma.

Uses the BCLC framework for treatment allocation, aligning North American and European practice on the same staging language.

How it compares

BCLC Staging vs Milan criteria

Different questions — BCLC allocates treatment across all stages, while Milan answers only whether a patient is within transplant criteria.

The Milan criteria (a single lesion ≤ 5 cm, or up to three lesions each ≤ 3 cm, without vascular invasion or extrahepatic spread) define transplant eligibility and are applied within BCLC stages A and B rather than in competition with them. A patient can be BCLC stage B and still within Milan, which is exactly the situation where transplantation is considered ahead of chemoembolisation.

Open the Milan criteria calculator →Mazzaferro V, Regalia E, Doci R, et al. Liver transplantation for the treatment of small hepatocellular carcinomas in patients with cirrhosis. N Engl J Med. 1996;334(11):693-699.

BCLC Staging vs Child-Pugh score

Child-Pugh is an input to BCLC, not an alternative to it — it supplies the liver function axis.

BCLC uses preserved (Child-Pugh A or B) versus end-stage (Child-Pugh C) function as one of its three axes, so the two are nested rather than competing. Child-Pugh alone says nothing about tumour burden or performance status and cannot allocate treatment in hepatocellular carcinoma.

Open the Child-Pugh score calculator →

BCLC Staging vs LI-RADS v2018

Sequential — LI-RADS establishes that a lesion is hepatocellular carcinoma, and BCLC then stages the confirmed disease.

LI-RADS categorises an observation on CT or MRI by its probability of being HCC, with LR-5 permitting a non-invasive diagnosis without biopsy. BCLC presupposes that diagnosis. Applying BCLC to an LR-3 observation stages something that has not yet been shown to be cancer.

Open the LI-RADS v2018 calculator →

BCLC Staging vs ALBI grade

Complementary — ALBI grades liver function more granularly than the binary preserved/end-stage split BCLC uses.

ALBI uses only bilirubin and albumin, avoiding the subjective ascites and encephalopathy items in Child-Pugh, and separates patients within Child-Pugh A who have meaningfully different reserve. Several groups use ALBI alongside BCLC to refine treatment selection within a stage, particularly when deciding between resection and less invasive options.

Open the ALBI grade calculator →

Pearls & pitfalls

  • Assess performance status and liver function BEFORE tumour burden. A 1 cm nodule in a Child-Pugh C patient is stage D, not stage 0.
  • A single nodule is stage 0 or A by size however large. An 8 cm solitary tumour with PS 0 and preserved function is stage A — size alone never creates stage B.
  • The two size boundaries are not interchangeable: 2 cm separates 0 from A in a solitary nodule; 3 cm separates A from B when there are 2 or 3.
  • Stage B means more than 3 nodules, or 2–3 nodules with at least one above 3 cm. The 2026 update added this wording precisely because 'multifocal' was being read inconsistently.
  • Performance status 1 is enough for stage C. Clinicians routinely under-score PS in patients they know well, and that single point changes the treatment pathway entirely.
  • Distinguish tumour thrombus from bland portal vein thrombosis before assigning stage C — they look similar on non-contrast imaging and mean different things.
  • Stage D driven by liver function alone is not necessarily terminal. Assess transplant candidacy before accepting that designation.
  • Treatment-stage migration is part of the system, not a departure from it: a patient may reasonably receive a treatment allocated to another stage when the recommended option is unsuitable.
  • BCLC applies to hepatocellular carcinoma only. Intrahepatic cholangiocarcinoma and liver metastases are staged differently.

Critical actions

  • Confirm the diagnosis meets non-invasive imaging criteria (LI-RADS 5 or equivalent) or obtain histology before assigning a stage and committing to treatment.
  • Score performance status honestly and, where possible, independently — it is the input most vulnerable to optimism.
  • Establish liver function formally with Child-Pugh, and consider portal pressure where resection is contemplated.
  • Refer every case to a hepatobiliary multidisciplinary meeting; BCLC frames the discussion rather than replacing it.
  • At stages 0 and A, consider resection, ablation and transplantation together rather than in sequence.
  • At stage D driven by liver function, assess transplant candidacy explicitly before defaulting to supportive care.
  • Re-stage at each reassessment — BCLC stage is not fixed, and both progression and improvement in liver function move it.

Why this score exists

The defining decision was to refuse to stage the tumour in isolation. Every other cancer staging system in wide use describes anatomy and leaves treatment selection to a separate step; BCLC folds liver function and performance status into the stage itself, on the argument that in a cancer arising in a failing organ those are not comorbidities but constraints on what the disease actually is. That is why a 1 cm tumour in a Child-Pugh C patient is stage D — a designation that looks harsh until you notice it is telling you something true, which is that no oncological treatment will help that person and only a new liver might. The trade-off, and the standing criticism of the system, is that bundling three axes into one label loses information, which is what the 2022 sub-stratification of stage B and the 2026 CUSE framework are both attempts to give back.

About the creator

  • María Reig

    First author, BCLC 2022 and 2026 updates; BCLC Group, Hospital Clínic Barcelona

    Led the two most recent revisions of the BCLC strategy.

  • Alejandro Forner

    Co-author of the 2022 and 2026 updates and of much of the underlying BCLC evidence base.

  • Jordi Bruix

    Originator of the BCLC staging system

    Developed the original BCLC framework linking stage to treatment allocation and led its successive revisions.

Limitations

  • Bundles three distinct axes into a single label, so patients with the same stage can differ substantially — the reason stage B was sub-stratified in 2022 and the CUSE framework added in 2026.
  • Stage C is heterogeneous, spanning a patient with PS 1 and a single nodule and one with extensive portal invasion and extrahepatic disease.
  • Performance status is subjectively assessed and systematically under-scored by clinicians who know the patient, which shifts patients out of stage C incorrectly.
  • The binary preserved/end-stage liver function split is coarse; Child-Pugh A and B patients are grouped together despite meaningfully different tolerance of treatment.
  • Treatment recommendations evolve faster than stage definitions, so a stage assigned some years ago may carry an outdated management implication even though the stage itself is unchanged.
  • Applies only to hepatocellular carcinoma, and not to intrahepatic cholangiocarcinoma, mixed tumours or metastatic disease.
  • Does not account for tumour biology or biomarkers such as alpha-fetoprotein, which independently affect prognosis and transplant outcomes.

If you are the patient

BCLC is the system doctors use to describe how advanced liver cancer is and, importantly, what treatment is likely to help. Unlike staging systems for most other cancers, it does not look only at the tumour. It considers three things together: how big and how many the tumours are, how well the liver itself is working, and how well the person is in general. That is because liver cancer usually develops in a liver already damaged by cirrhosis, and the health of the rest of the liver decides what treatment is safely possible. The stages run from 0 (very early, a single small tumour, often curable) through A and B, to C (advanced) and D. Stage D usually means treatment is aimed at comfort rather than cure — but there is an important exception: if someone is in stage D mainly because their liver is failing rather than because the cancer is extensive, a liver transplant may still be an option, and that is always worth asking about.

Frequently asked questions

What is BCLC staging?#

The Barcelona Clinic Liver Cancer system, which stages hepatocellular carcinoma as 0, A, B, C or D using three axes together — tumour burden, liver function and performance status — and links each stage to a treatment recommendation.

What are the BCLC stage definitions?#

Stage 0 is a single nodule of 2 cm or less with PS 0 and preserved liver function. Stage A is a single larger nodule or 2–3 nodules all 3 cm or less. Stage B is multifocal disease — more than 3 nodules, or 2–3 with at least one above 3 cm. Stage C is portal invasion, extrahepatic spread or PS 1–2. Stage D is PS 3–4 or end-stage liver function.

What changed in the BCLC 2026 update?#

The stage definitions are unchanged from 2022. The update explicitly defines multifocal disease in stage B as more than 3 nodules or 2–3 nodules with at least one above 3 cm, adds stereotactic body radiotherapy and transarterial radioembolisation as potentially curative options at stages 0 and A, consolidates combination immunotherapy as preferred first line at stage C, and introduces the CUSE decision framework.

Is a large single tumour stage B?#

No. A single nodule is stage 0 or A by size alone, however large — an 8 cm solitary tumour with PS 0 and preserved liver function is stage A. Stage B requires multifocal disease: more than 3 nodules, or 2–3 nodules with at least one above 3 cm.

Why is a small tumour sometimes stage D?#

Because performance status and liver function are assessed before tumour burden. A 1 cm nodule in a patient with Child-Pugh C cirrhosis or ECOG 3 is stage D, since no oncological treatment will help. Where the stage is driven by liver function alone, transplantation may still be an option.

Does BCLC replace TNM staging in liver cancer?#

In practice, yes, for treatment allocation. TNM stages the tumour anatomically but does not account for liver function or performance status, both of which determine what treatment is possible in a cancer arising in a cirrhotic liver. Both EASL and AASLD build their guidance on BCLC.

What is treatment-stage migration?#

The principle, built into BCLC, that a patient may receive a treatment allocated to a different stage when the recommended option is unsuitable — for example moving to the next-best option when resection is precluded by portal hypertension. It is part of the system rather than a departure from it.

Can a patient move between BCLC stages?#

Yes, in both directions. Tumour progression moves a patient up, and improvement in liver function or performance status can move them down — which is why re-staging at each reassessment matters, and why treating a decompensation can change what is oncologically possible.

Related calculators

  • Milan Criteria — Liver transplant eligibility in hepatocellular carcinoma
  • LI-RADS v2018 (CT/MRI) — Liver observation category from size, APHE and major features
  • Child-Pugh Score — Assesses the prognosis of chronic liver disease, mainly cirrhosis
  • ALBI Grade — Albumin-bilirubin liver function grade in HCC
  • MELD-Na — Assesses the severity of chronic liver disease
  • Baveno VII Criteria — cACLD, CSPH and sparing screening endoscopy
  • Metroticket 2.0 — AFP-adjusted up-to-seven for HCC transplant eligibility
  • GALAD Score — HCC detection from gender, age, AFP-L3, AFP and DCP

References

Original / primary reference

  1. Reig M, Forner A, Ávila MA, et al. BCLC strategy for prognosis prediction and treatment recommendations: The 2026 update. J Hepatol. 2026;84(3):631-654.
  2. Reig M, Forner A, Rimola J, et al. BCLC strategy for prognosis prediction and treatment recommendation: The 2022 update. J Hepatol. 2022;76(3):681-693.

Guidelines

  1. European Association for the Study of the Liver. EASL Clinical Practice Guidelines: Management of hepatocellular carcinoma. J Hepatol. 2018;69(1):182-236.
  2. Marrero JA, Kulik LM, Sirlin CB, et al. Diagnosis, Staging, and Management of Hepatocellular Carcinoma: 2018 Practice Guidance by the American Association for the Study of Liver Diseases. Hepatology. 2018;68(2):723-750.

Last updated August 1, 2026. Clinical knowledge base written and curated by GastroAGI Team from primary medical literature.

Written from primary literature and not yet independently clinically reviewed.

For use by qualified healthcare professionals. This calculator supports clinical judgement and does not replace it.