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Most used

21
MELD-NaAssesses the severity of chronic liver diseaseChild-Pugh ScoreAssesses the prognosis of chronic liver disease, mainly cirrhosisFIB-4 IndexLiver fibrosis scoring indexAPRIAST to platelet ratio — liver fibrosisMaddrey's DFAlcoholic hepatitis severityGlasgow-BlatchfordUpper GI bleed risk stratificationGAHSGlasgow alcoholic hepatitis scoreMontreal IBDIBD classification — CD & UCMayo ScoreUlcerative colitis activityBISAP ScoreBedside index for severity of pancreatitisCLIF-SOFAOrgan failure scoring in cirrhosisAlcohol ContentStandard drinks & alcohol grams calculatorAARC-ACLFAcute-on-chronic liver failure gradePELD / CR ScorePediatric end-stage liver diseaseHarvey-BradshawCrohn's disease activity indexCTSICT severity index — pancreatitisRockall ScoreGI bleed rebleeding & mortality riskCAGEAlcohol use disorder screening (4 questions)MELD 3.0Updated MELD — sex-inclusive formulaAUDIT ScoreAlcohol use disorders identification testVOCAL-Penn ScorePost-operative mortality risk in cirrhosis surgery

Liver & Cirrhosis

17
ALBI GradeAlbumin-bilirubin liver function grade in HCCUKELD ScoreUK model for end-stage liver diseaseMELD-XIMELD excluding INR — for anticoagulated patientsWest Haven CriteriaHepatic encephalopathy gradingMilan CriteriaLiver transplant eligibility in hepatocellular carcinomaLI-RADS v2018 (CT/MRI)Liver observation category from size, APHE and major featuresBCLC StagingHepatocellular carcinoma stage and treatment allocationSimplified AIH CriteriaSimplified criteria for autoimmune hepatitisRevised Original AIH ScoreIAIHG 1999 comprehensive autoimmune hepatitis scoreSAAGSerum-ascites albumin gradient — cause of ascitesR FactorHepatocellular vs cholestatic pattern in liver injuryCLIF-C ACLFMortality prediction in acute-on-chronic liver failureKing's College CriteriaTransplant criteria in acute liver failureGALAD ScoreHCC detection from gender, age, AFP-L3, AFP and DCPMetroticket 2.0AFP-adjusted up-to-seven for HCC transplant eligibilityRUCAMCausality in drug- and herb-induced liver injuryBaveno VII CriteriacACLD, CSPH and sparing screening endoscopy

Fibrosis & MASLD

8
NAFLD Fibrosis ScoreAdvanced fibrosis probability in MASLD/NAFLDBARD ScoreBMI, AST/ALT ratio, diabetes — MASLD fibrosisFatty Liver IndexPredicts hepatic steatosis from routine labsFibrotic NASH Index (FNI)At-risk NASH probability from AST, HbA1c and HDLNAFLD Activity Score (NAS)Histologic activity grade — steatosis, inflammation, ballooningMEFIB IndexMRE + FIB-4 rule for significant fibrosis (≥F2) in MASLDFAST ScoreFibroScan-AST — at-risk NASH from LSM, CAP and ASTSAFE ScoreSteatosis-Associated Fibrosis Estimator for MASLD in primary care

Pancreas & Biliary

8
Ranson's CriteriaAcute pancreatitis severity at 48 hoursGlasgow-Imrie CriteriaAcute pancreatitis severity — the PANCREAS criteriaHAPSHarmless acute pancreatitis scoreTokyo Guidelines — CholangitisTG18 diagnosis and severity grade for acute cholangitisTokyo Guidelines — CholecystitisTG18 diagnosis and severity grade for acute cholecystitisBiliary Pain (Rome IV)Rome IV — defining biliary-type pain before interventionFunctional Pancreatic SODRome IV — pancreatic sphincter of Oddi disorderRevised Atlanta ClassificationAcute pancreatitis severity — mild, moderately severe, severe

IBD

9
Truelove & Witts CriteriaAcute severe ulcerative colitis — admission decisionUCEISUlcerative colitis endoscopic index of severitySCCAISimple clinical colitis activity index — symptoms onlyCDAICrohn's disease activity index — the trial standardSES-CDEndoscopic severity in Crohn's diseasePUCAIPaediatric ulcerative colitis activity indexTravis (Oxford) CriteriaDay 3 colectomy risk in acute severe ulcerative colitisHo IndexDay 3 steroid failure risk in acute severe ulcerative colitisRutgeerts ScorePostoperative Crohn's recurrence at ileocolonoscopy

GI Bleeding

7
EVendo ScorePredicts oesophageal varices needing treatmentForrest ClassificationPeptic ulcer bleeding — rebleeding risk at endoscopyAIMS65 ScoreUpper GI bleed mortality — five bedside criteriaOakland ScoreSafe-discharge risk for acute lower GI bleedingABC ScoreAge, blood tests, comorbidities — GI bleed mortalitySarin ClassificationEndoscopic classification of gastric varicesEGUS (Gastric Ulcer)Malignancy risk in a gastric ulcer, and who needs repeat endoscopy

Alcohol

2
ABIC ScoreAge, bilirubin, INR, creatinine — alcoholic hepatitisLille ModelSteroid response at day 7 in alcoholic hepatitis

Upper GI

4
Chicago Classification v4.0Oesophageal motility pattern from high-resolution manometryLA Classification (Oesophagitis)Los Angeles grade A–D for erosive oesophagitisPrague C & M CriteriaCircumferential and maximal extent of Barrett's oesophagusEREFS (Eosinophilic Oesophagitis)Endoscopic reference score — oedema, rings, exudates, furrows, stricture

Colorectal

3
Boston Bowel Prep ScaleColonoscopy preparation adequacy by segmentStool Osmotic GapOsmotic vs secretory diarrhoea from stool electrolytesATLAS Score (C. difficile)Predicted response to therapy in Clostridioides difficile infection

Functional GI

37
Bristol Stool ScaleStool form types 1–7 and colonic transitRome IV Criteria for IBSIrritable bowel syndrome diagnosis and subtypeFunctional ConstipationRome IV — two of six items, IBS excludedOpioid-Induced ConstipationRome IV — constipation tied to opioid therapyFunctional DiarrhoeaRome IV — loose stools without predominant painFunctional Bloating / DistensionRome IV — bloating without other bowel disorder criteriaUnspecified Functional Bowel DisorderRome IV — bowel symptoms fitting no other categoryCentrally Mediated Abdominal Pain (CAPS)Rome IV — continuous pain unrelated to gut eventsNarcotic Bowel SyndromeRome IV — opioid-induced hyperalgesia of the gutFaecal Incontinence (Rome IV)Rome IV — the criteria, and why nobody is askedFunctional Anorectal PainLevator ani, unspecified pain and proctalgia fugaxFunctional Defecation DisordersRome IV — dyssynergia and inadequate propulsionInfant RegurgitationRome IV — the happy spitter, and the alarm features that rule it outInfant ColicRome IV — recurrent unexplained crying in a well infant under 5 monthsInfant DyscheziaRome IV — straining before a soft stool, and why not to intervenePaediatric Functional ConstipationRome IV — two of six over one month, with overflow soiling as a criterionToddler's DiarrhoeaRome IV functional diarrhoea of childhood — painless, thriving childPaediatric Cyclic Vomiting SyndromeRome IV — both age bands, with different criteria for eachPaediatric Rumination SyndromeRome IV — infant and child/adolescent criteriaFunctional Nausea & Vomiting (Children)Rome IV — two separate disorders that can be met togetherAerophagiaRome IV — distension that increases through the dayPaediatric Functional DyspepsiaRome IV — four times a month, with PDS and EPS subtypingPaediatric Irritable Bowel SyndromeRome IV — plus the constipation clause clinicians missAbdominal MigraineRome IV — stereotypical incapacitating episodes weeks apartFunctional Abdominal Pain — NOSRome IV — the residual category, reached after the other threeNonretentive Faecal IncontinenceRome IV — soiling without retention, where laxatives make it worseFunctional DyspepsiaRome IV — with PDS and EPS subtypingRumination SyndromeRome IV — effortless regurgitation without retchingCyclic Vomiting SyndromeRome IV — stereotypical episodic vomitingCannabinoid HyperemesisRome IV — CVS pattern relieved by cannabis cessationChronic Nausea & VomitingRome IV — chronic nausea and vomiting syndromeBelching DisordersRome IV — supragastric vs gastric belchingFunctional HeartburnRome IV — heartburn with normal acid exposureReflux HypersensitivityRome IV — normal acid exposure, positive symptom associationFunctional Chest PainRome IV — non-cardiac, non-reflux chest painGlobusRome IV — painless lump-in-throat sensationFunctional DysphagiaRome IV — dysphagia with normal endoscopy and manometry
  1. Calculators
  2. /
  3. MEFIB Index
Fibrosis & MASLD

MEFIB Index

MRE + FIB-4 rule for significant fibrosis (≥F2) in MASLD

Imaging

Magnetic resonance elastography.

Liver stiffness by 2D magnetic resonance elastography (60 Hz). The MEFIB threshold is 3.3 kPa.

FIB-4 components

Used to compute FIB-4 = (age × AST) ÷ (platelets × √ALT).

A dichotomous rule, not a continuous score. FIB-4 is computed from age, AST, ALT and platelets, then combined with the MR elastography liver stiffness.

When to use
Use it where MRE is available and the question is whether a patient with metabolic liver disease has significant fibrosis worth treating — most often to enrich a treatment or trial population with people genuinely at stage F2 or above, or to confidently exclude significant fibrosis when both markers are low. It is best thought of as the second step after a blood-based triage: FIB-4 is calculated for everyone, and those who reach MRE have the two combined. It is not a first-line population screen (MRE is expensive and not widely available), it does not stage fibrosis precisely, and it is validated in MASLD/NAFLD rather than in viral or alcohol-related disease.
Why use it
Because the single biggest problem in selecting patients for NASH fibrosis therapy is false positives: send everyone with a raised FIB-4 or a modestly stiff liver to biopsy or treatment and a large fraction turn out not to have significant fibrosis. MEFIB was built to solve exactly that. By requiring both an abnormal MRE and an abnormal FIB-4 it achieves a positive predictive value around 90% or better for ≥ F2 — substantially higher than either test alone — so a positive result can stand in for the biopsy that would otherwise be needed to confirm a treatment candidate. Its authors framed it explicitly as a tool for non-invasively identifying candidates for pharmacological treatment of NASH-related fibrosis.
Formula, evidence and interpretation

About the MEFIB Index (MRE + FIB-4)

The MEFIB index is a dichotomous rule that combines magnetic resonance elastography (MRE) liver stiffness with the blood-based FIB-4 to identify significant fibrosis (stage F2 or higher) in metabolic (MASLD/NAFLD) liver disease. It is positive when MRE is at least 3.3 kPa and FIB-4 is at least 1.6, and this combination carried a positive predictive value of 91–97% for ≥ F2 across the derivation and validation cohorts — high enough to select candidates for fibrosis-directed treatment or a trial without a biopsy. When both values fall below their thresholds the rule is negative, with a negative predictive value of 86–93%. A result where only one criterion is met is indeterminate.

On this page

  • Formula
  • Interpreting the result
  • Inputs
  • What it returns
  • How it is calculated
  • Facts & figures
  • Evidence
  • How it compares
  • Pearls & pitfalls
  • Critical actions
  • Why it exists
  • About the creator
  • Limitations
  • If you are the patient
  • FAQ
  • Related calculators
  • References

Formula

MEFIB-positive if (MRE ≥ 3.3 kPa) AND (FIB-4 ≥ 1.6). MEFIB-negative if both are below threshold. Otherwise indeterminate. [FIB-4 = (age × AST) ÷ (platelets × √ALT)]
MRE
MR elastography liver stiffness in kPa; threshold 3.3.
FIB-4
Computed from age (years), AST (U/L), ALT (U/L) and platelets (×10⁹/L); threshold 1.6.
  • MEFIB is a rule, not an equation — there is no continuous MEFIB number, only the three-way classification.
  • Both conditions must hold for a positive; a single abnormal marker is deliberately not enough, which is what gives the positive result its high specificity.
  • The FIB-4 sub-calculation uses the same conventional units as the standalone FIB-4: AST and ALT in U/L, platelets in ×10⁹/L.

Interpreting the result

Treat MEFIB as a high-confidence rule-in when positive and a reasonable rule-out when doubly negative. A positive result — both criteria met — makes significant fibrosis very likely (PPV 91–97%) and, in the context the score was built for, is strong enough to enrol a patient for fibrosis-directed therapy or a trial without histological confirmation. A doubly negative result makes significant fibrosis unlikely (NPV 86–93%) and supports continued metabolic management with reassessment over time; longitudinal data also link a MEFIB-negative result to a low rate of subsequent liver-related events. The discordant case, where only one marker is abnormal, is genuinely indeterminate and should prompt a further test rather than being forced into a positive or negative call.

ScoreBandWhat it meansAction
MRE ≥ 3.3 & FIB-4 ≥ 1.6MEFIB-positiveSignificant fibrosis (≥ F2) very likely — PPV 91–97% across cohortsSuitable to identify a candidate for fibrosis-directed treatment or trial; specialist referral
Discordant (one criterion met)IndeterminateMEFIB neither rules significant fibrosis in nor outFurther testing — MRI-PDFF, repeat assessment or biopsy where it changes management
MRE < 3.3 & FIB-4 < 1.6MEFIB-negativeSignificant fibrosis unlikely — NPV 86–93%; low rate of liver-related events on follow-upContinue metabolic management and reassess over time

What the MEFIB Index needs (2 inputs)

MRE liver stiffness (kPa)
Liver stiffness measured by 2D magnetic resonance elastography at 60 Hz. The MEFIB threshold is 3.3 kPa. MRE is the most accurate non-invasive stiffness measurement but requires an MRI scanner and specific software.
FIB-4 (from age, AST, ALT, platelets)
The FIB-4 index is computed internally as (age × AST) ÷ (platelets × √ALT); the MEFIB threshold is 1.6. Because age sits in the FIB-4 numerator, the same age-related caveats that affect FIB-4 carry into MEFIB.

What it returns

MEFIB result
Positive (both criteria met), negative (both below threshold) or indeterminate (discordant). It is a rule-in/rule-out classification, not a fibrosis stage.
Component values
The MRE stiffness and the computed FIB-4, each shown against its threshold, so it is clear which criterion is or is not met.

How it is calculated

MEFIB pairs a blood marker and an imaging marker that fail in different ways, so that requiring both to be abnormal removes most false positives. FIB-4 is cheap and sensitive but not specific — it is raised by age and by many non-fibrotic causes. MRE is highly accurate but, applied alone at a low threshold, still labels some people without significant fibrosis. The MEFIB authors searched combinations of the two in a biopsied MASLD cohort and found that the pairing of MRE ≥ 3.3 kPa with FIB-4 ≥ 1.6 maximised the positive predictive value for stage ≥ F2. The logic is conjunctive rather than additive: neither marker is weighted or summed, they simply both have to clear their cut-off for the rule to fire.

Facts & figures

Performance of the MEFIB rule for ≥ F2
CohortMetricValue
UCSD-NAFLD (derivation)PPV of a positive rule97.1%
UCSD-NAFLD (derivation)AUROC0.90 (0.85–0.95)
Japan-NAFLD (validation)PPV of a positive rule91.0%
Japan-NAFLD (validation)AUROC0.84 (0.78–0.89)
Both cohortsNPV of a doubly negative rule86–93%

Target is significant fibrosis (stage F2 or higher), not advanced fibrosis alone.

The two thresholds
MarkerThresholdType
MRE liver stiffness≥ 3.3 kPaImaging (MRI-based)
FIB-4≥ 1.6Blood (age, AST, ALT, platelets)

Both must be met for a positive result; both must be below for a negative result.

Evidence

Derivation — UCSD-NAFLD cohort

2021

A prospective, biopsy-proven MASLD cohort at UC San Diego with contemporaneous MRE and FIB-4. Combinations of MRE and FIB-4 thresholds were tested against biopsy fibrosis stage, and MRE ≥ 3.3 kPa with FIB-4 ≥ 1.6 was selected as the rule maximising positive predictive value for stage ≥ F2.

For ≥ F2, the positive rule had a positive predictive value of 97.1% and an AUROC of 0.90 (95% CI 0.85–0.95).

External validation — Japan-NAFLD cohort

2021

An independent biopsy-proven MASLD cohort in Japan, applying the same MRE ≥ 3.3 kPa and FIB-4 ≥ 1.6 rule.

Positive predictive value 91.0% for ≥ F2, AUROC 0.84 (95% CI 0.78–0.89) — confirming the rule transports across populations.

Head-to-head against FAST

2022

Tamaki and colleagues compared MEFIB with the FibroScan-AST (FAST) score for identifying candidates for pharmacological treatment of NASH-related fibrosis in paired cohorts.

MEFIB identified treatment candidates with a higher positive predictive value than FAST, at the cost of requiring MRE rather than the more widely available FibroScan.

How it compares

MEFIB Index vs FIB-4 alone

MEFIB is FIB-4 plus MRE — use FIB-4 alone as the cheap first-line rule-out, and add MRE to form MEFIB when you need a high-confidence rule-in for significant fibrosis without biopsy.

FIB-4 on its own is sensitive but not specific: many raised results are not significant fibrosis, especially in older patients. Adding the requirement of an abnormal MRE lifts the positive predictive value for ≥ F2 from FIB-4's modest figure to around 90% or more. The trade-off is access and cost — FIB-4 is free and universal, while MEFIB needs an MRI-based scan — so the natural pathway is FIB-4 for everyone and MEFIB for those in whom a confident rule-in would change management.

Open the FIB-4 alone calculator →

MEFIB Index vs FAST score

Both select candidates for NASH fibrosis therapy, but MEFIB uses MRE and reached a higher positive predictive value head-to-head, while FAST uses the cheaper, more available FibroScan — accuracy versus access.

FAST combines FibroScan liver stiffness and CAP with AST to flag at-risk NASH, whereas MEFIB combines MRE with FIB-4 to flag significant fibrosis. In a direct comparison MEFIB identified treatment candidates with a higher positive predictive value than FAST, reflecting MRE's superior accuracy; but FibroScan is far more widely available than MRE, so FAST is the more deployable of the two where MRI capacity is limited.

Open the FAST score calculator →

MEFIB Index vs MAST score

Both are MRI-based, but they target different things — MAST estimates at-risk NASH (activity plus fibrosis) while MEFIB rules significant fibrosis in or out — so the choice follows whether the question is disease activity or fibrosis stage.

MAST folds MRI-PDFF, MRE and AST into a continuous probability of at-risk NASH (NAS ≥ 4 with F ≥ 2). MEFIB uses MRE and FIB-4 as a dichotomous rule for significant fibrosis (≥ F2) regardless of activity. Both rely on MRE, so availability is similar; the distinction is conceptual — an activity-and-fibrosis composite versus a pure fibrosis-stage rule.

Pearls & pitfalls

  • MEFIB is a conjunction: both MRE ≥ 3.3 kPa and FIB-4 ≥ 1.6 must hold. A single abnormal marker is indeterminate, not positive.
  • Its strength is the positive result. A MEFIB-positive is a high-confidence rule-in for ≥ F2; treat the negative result as a reasonable but less absolute rule-out.
  • FIB-4's age dependence carries over. In older patients a raised FIB-4 may reflect age rather than fibrosis, so the MRE half does more of the work.
  • It needs MRE, which requires an MRI scanner with elastography software, adds cost, and is unavailable to patients with common MRI contraindications.
  • The target is significant fibrosis (≥ F2), not advanced fibrosis (≥ F3) — do not read a positive result as cirrhosis.
  • It is validated in MASLD/NAFLD. Do not apply the 3.3 kPa and 1.6 thresholds to viral or alcohol-related liver disease without separate evidence.
  • The discordant group is real and common; resist the urge to round it up to positive or down to negative.

Critical actions

  • Confirm both thresholds explicitly before acting — a positive requires MRE ≥ 3.3 kPa and FIB-4 ≥ 1.6 together.
  • On a positive result, proceed to specialist assessment and consider fibrosis-directed treatment or trial eligibility; biopsy is often avoidable given the high PPV.
  • On a doubly negative result, continue metabolic management and set an interval for reassessment rather than discharging.
  • On a discordant result, arrange a further test (MRI-PDFF, repeat MRE/FIB-4, or biopsy) rather than committing to a treatment decision.
  • Interpret FIB-4 with its usual caveats — check for non-hepatic causes of thrombocytopenia or a raised AST before relying on the blood half.

Why this score exists

The MEFIB authors were solving a specific operational problem: how to select patients with genuine stage ≥ F2 fibrosis for NASH drug trials without biopsying everyone. Their design choice — demand that both an accurate imaging marker and a cheap blood marker be abnormal — was aimed squarely at the positive predictive value, because in a trial-enrichment setting a false positive is expensive and a missed borderline case is tolerable. That framing explains the rule's shape and its limits: it is optimised to be right when it says 'yes', it accepts an indeterminate middle group rather than forcing a call, and it was never intended as a population screen, because its imaging half is not something you can run at scale.

About the creator

  • Jinho Jung

    First author, 2021 derivation study

    Derived the MEFIB index, pairing MR elastography with FIB-4 to rule in significant fibrosis.

  • Rohit Loomba

    Senior author

    Led the San Diego MASLD imaging programme behind MEFIB and much of the comparative non-invasive-test literature.

Limitations

  • It requires MRE, which is expensive, not widely available, and precluded by common MRI contraindications, so it cannot serve as a first-line or population test.
  • It is a rule for significant fibrosis (≥ F2), not a fibrosis stager and not specific to advanced fibrosis or cirrhosis.
  • The blood half inherits FIB-4's weaknesses, including its rise with age and its distortion by non-hepatic causes of thrombocytopenia or transaminitis.
  • A substantial discordant group falls into the indeterminate zone, where the rule provides no answer.
  • It was derived and validated in MASLD/NAFLD and should not be transferred to other liver diseases without dedicated evidence.
  • The high positive predictive value reflects cohorts with a meaningful prevalence of significant fibrosis; in a very low-prevalence setting the predictive values will differ.

If you are the patient

MEFIB is a way of combining two different liver tests to get a more confident answer about scarring. One is a special MRI scan of the liver (called MR elastography, or MRE) that measures how stiff the liver is. The other is FIB-4, a score worked out from your age and three routine blood results. On their own, each test can suggest scarring when there isn't much. MEFIB only calls the result 'positive' when both tests agree that the liver is abnormal — and when they do agree, significant scarring is very likely (right about 9 times out of 10), which can spare you a liver biopsy. If both tests are normal, significant scarring is unlikely. If the two disagree, the result is 'in between' and your team will usually arrange another test. The main catch is that MEFIB needs the special MRI scan, which isn't available everywhere, so it is used as a careful second step rather than a first screening test.

Frequently asked questions

What is the MEFIB index?#

MEFIB is a combined test for significant liver fibrosis (stage F2 or higher) in metabolic fatty liver disease. It is positive when MR elastography liver stiffness is at least 3.3 kPa and the blood-based FIB-4 is at least 1.6. Requiring both to be abnormal gives a high positive predictive value — around 91–97% — so a positive result can identify a treatment candidate without a biopsy.

What do the MEFIB thresholds mean?#

MEFIB-positive means both MRE ≥ 3.3 kPa and FIB-4 ≥ 1.6, indicating significant fibrosis is very likely. MEFIB-negative means both are below those cut-offs, indicating it is unlikely. If only one criterion is met the result is indeterminate, and a further test is needed. The two thresholds were chosen to maximise the positive predictive value for stage ≥ F2.

How accurate is MEFIB?#

In the UC San Diego derivation cohort a positive MEFIB had a positive predictive value of 97.1% for significant fibrosis, with an AUROC of 0.90; in an independent Japanese validation cohort the positive predictive value was 91.0% with an AUROC of 0.84. A doubly negative result had a negative predictive value of 86–93%.

Is MEFIB better than the FAST score?#

In a head-to-head comparison, MEFIB identified candidates for NASH fibrosis treatment with a higher positive predictive value than FAST, reflecting the greater accuracy of MR elastography. However, FAST uses FibroScan, which is far more widely available and cheaper than the MRI-based MRE that MEFIB requires, so FAST is more practical where MRI capacity is limited.

Does MEFIB replace a liver biopsy?#

For confirming a treatment candidate it often can. The whole purpose of MEFIB was to identify people with stage ≥ F2 fibrosis non-invasively, and the ~90%+ positive predictive value of a positive result is high enough that many patients no longer need biopsy. Indeterminate and some negative results may still require further testing, and biopsy remains an option where it would change management.

Which fibrosis stage does MEFIB detect?#

Significant fibrosis, meaning stage F2 or higher on the standard 0–4 scale. That is different from advanced fibrosis (F3–F4), so a positive MEFIB should not be read as cirrhosis; it indicates fibrosis meaningful enough to warrant treatment consideration.

Related calculators

  • FIB-4 Index — Liver fibrosis scoring index
  • Fibrotic NASH Index (FNI) — At-risk NASH probability from AST, HbA1c and HDL
  • NAFLD Fibrosis Score — Advanced fibrosis probability in MASLD/NAFLD
  • APRI — AST to platelet ratio — liver fibrosis
  • FAST Score — FibroScan-AST — at-risk NASH from LSM, CAP and AST
  • SAFE Score — Steatosis-Associated Fibrosis Estimator for MASLD in primary care

References

Original / primary reference

  1. Jung J, Loomba RR, Imajo K, et al. MRE combined with FIB-4 (MEFIB) index in detection of candidates for pharmacological treatment of NASH-related fibrosis. Gut. 2021;70(10):1946-1953.

Validation

  1. Tamaki N, Imajo K, Sharpton S, et al. Magnetic resonance elastography plus Fibrosis-4 versus FibroScan-aspartate aminotransferase in detection of candidates for pharmacological treatment of NASH-related fibrosis. Hepatology. 2022;75(3):661-672.

Component tests

  1. Loomba R, Wolfson T, Ang B, et al. Magnetic resonance elastography predicts advanced fibrosis in patients with nonalcoholic fatty liver disease: a prospective study. Hepatology. 2014;60(6):1920-1928.
  2. Sterling RK, Lissen E, Clumeck N, et al. Development of a simple noninvasive index to predict significant fibrosis in patients with HIV/HCV coinfection (FIB-4). Hepatology. 2006;43(6):1317-1325.

Clinical practice guidelines

  1. Berzigotti A, Tsochatzis E, Boursier J, et al. EASL Clinical Practice Guidelines on non-invasive tests for evaluation of liver disease severity and prognosis – 2021 update. J Hepatol. 2021;75(3):659-689.
  2. EASL-EASD-EASO Clinical Practice Guidelines on the management of metabolic dysfunction-associated steatotic liver disease (MASLD). J Hepatol. 2024;81(3):492-542.
  3. Rinella ME, Neuschwander-Tetri BA, Siddiqui MS, et al. AASLD Practice Guidance on the clinical assessment and management of nonalcoholic fatty liver disease. Hepatology. 2023;77(5):1797-1835.

Further reading

  1. Tavaglione F, De Vincentis A, Jamialahmadi O, et al. Development and Validation of a Score for Fibrotic Nonalcoholic Steatohepatitis (FNI). Clin Gastroenterol Hepatol. 2023;21(6):1523-1532.e1.

Last updated July 30, 2026. Clinical knowledge base written and curated by GastroAGI Team from primary medical literature.

Written from primary literature and not yet independently clinically reviewed.

For use by qualified healthcare professionals. This calculator supports clinical judgement and does not replace it.