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116 calculators match

Most used

21
MELD-NaAssesses the severity of chronic liver diseaseChild-Pugh ScoreAssesses the prognosis of chronic liver disease, mainly cirrhosisFIB-4 IndexLiver fibrosis scoring indexAPRIAST to platelet ratio — liver fibrosisMaddrey's DFAlcoholic hepatitis severityGlasgow-BlatchfordUpper GI bleed risk stratificationGAHSGlasgow alcoholic hepatitis scoreMontreal IBDIBD classification — CD & UCMayo ScoreUlcerative colitis activityBISAP ScoreBedside index for severity of pancreatitisCLIF-SOFAOrgan failure scoring in cirrhosisAlcohol ContentStandard drinks & alcohol grams calculatorAARC-ACLFAcute-on-chronic liver failure gradePELD / CR ScorePediatric end-stage liver diseaseHarvey-BradshawCrohn's disease activity indexCTSICT severity index — pancreatitisRockall ScoreGI bleed rebleeding & mortality riskCAGEAlcohol use disorder screening (4 questions)MELD 3.0Updated MELD — sex-inclusive formulaAUDIT ScoreAlcohol use disorders identification testVOCAL-Penn ScorePost-operative mortality risk in cirrhosis surgery

Liver & Cirrhosis

17
ALBI GradeAlbumin-bilirubin liver function grade in HCCUKELD ScoreUK model for end-stage liver diseaseMELD-XIMELD excluding INR — for anticoagulated patientsWest Haven CriteriaHepatic encephalopathy gradingMilan CriteriaLiver transplant eligibility in hepatocellular carcinomaLI-RADS v2018 (CT/MRI)Liver observation category from size, APHE and major featuresBCLC StagingHepatocellular carcinoma stage and treatment allocationSimplified AIH CriteriaSimplified criteria for autoimmune hepatitisRevised Original AIH ScoreIAIHG 1999 comprehensive autoimmune hepatitis scoreSAAGSerum-ascites albumin gradient — cause of ascitesR FactorHepatocellular vs cholestatic pattern in liver injuryCLIF-C ACLFMortality prediction in acute-on-chronic liver failureKing's College CriteriaTransplant criteria in acute liver failureGALAD ScoreHCC detection from gender, age, AFP-L3, AFP and DCPMetroticket 2.0AFP-adjusted up-to-seven for HCC transplant eligibilityRUCAMCausality in drug- and herb-induced liver injuryBaveno VII CriteriacACLD, CSPH and sparing screening endoscopy

Fibrosis & MASLD

8
NAFLD Fibrosis ScoreAdvanced fibrosis probability in MASLD/NAFLDBARD ScoreBMI, AST/ALT ratio, diabetes — MASLD fibrosisFatty Liver IndexPredicts hepatic steatosis from routine labsFibrotic NASH Index (FNI)At-risk NASH probability from AST, HbA1c and HDLNAFLD Activity Score (NAS)Histologic activity grade — steatosis, inflammation, ballooningMEFIB IndexMRE + FIB-4 rule for significant fibrosis (≥F2) in MASLDFAST ScoreFibroScan-AST — at-risk NASH from LSM, CAP and ASTSAFE ScoreSteatosis-Associated Fibrosis Estimator for MASLD in primary care

Pancreas & Biliary

8
Ranson's CriteriaAcute pancreatitis severity at 48 hoursGlasgow-Imrie CriteriaAcute pancreatitis severity — the PANCREAS criteriaHAPSHarmless acute pancreatitis scoreTokyo Guidelines — CholangitisTG18 diagnosis and severity grade for acute cholangitisTokyo Guidelines — CholecystitisTG18 diagnosis and severity grade for acute cholecystitisBiliary Pain (Rome IV)Rome IV — defining biliary-type pain before interventionFunctional Pancreatic SODRome IV — pancreatic sphincter of Oddi disorderRevised Atlanta ClassificationAcute pancreatitis severity — mild, moderately severe, severe

IBD

9
Truelove & Witts CriteriaAcute severe ulcerative colitis — admission decisionUCEISUlcerative colitis endoscopic index of severitySCCAISimple clinical colitis activity index — symptoms onlyCDAICrohn's disease activity index — the trial standardSES-CDEndoscopic severity in Crohn's diseasePUCAIPaediatric ulcerative colitis activity indexTravis (Oxford) CriteriaDay 3 colectomy risk in acute severe ulcerative colitisHo IndexDay 3 steroid failure risk in acute severe ulcerative colitisRutgeerts ScorePostoperative Crohn's recurrence at ileocolonoscopy

GI Bleeding

7
EVendo ScorePredicts oesophageal varices needing treatmentForrest ClassificationPeptic ulcer bleeding — rebleeding risk at endoscopyAIMS65 ScoreUpper GI bleed mortality — five bedside criteriaOakland ScoreSafe-discharge risk for acute lower GI bleedingABC ScoreAge, blood tests, comorbidities — GI bleed mortalitySarin ClassificationEndoscopic classification of gastric varicesEGUS (Gastric Ulcer)Malignancy risk in a gastric ulcer, and who needs repeat endoscopy

Alcohol

2
ABIC ScoreAge, bilirubin, INR, creatinine — alcoholic hepatitisLille ModelSteroid response at day 7 in alcoholic hepatitis

Upper GI

4
Chicago Classification v4.0Oesophageal motility pattern from high-resolution manometryLA Classification (Oesophagitis)Los Angeles grade A–D for erosive oesophagitisPrague C & M CriteriaCircumferential and maximal extent of Barrett's oesophagusEREFS (Eosinophilic Oesophagitis)Endoscopic reference score — oedema, rings, exudates, furrows, stricture

Colorectal

3
Boston Bowel Prep ScaleColonoscopy preparation adequacy by segmentStool Osmotic GapOsmotic vs secretory diarrhoea from stool electrolytesATLAS Score (C. difficile)Predicted response to therapy in Clostridioides difficile infection

Functional GI

37
Bristol Stool ScaleStool form types 1–7 and colonic transitRome IV Criteria for IBSIrritable bowel syndrome diagnosis and subtypeFunctional ConstipationRome IV — two of six items, IBS excludedOpioid-Induced ConstipationRome IV — constipation tied to opioid therapyFunctional DiarrhoeaRome IV — loose stools without predominant painFunctional Bloating / DistensionRome IV — bloating without other bowel disorder criteriaUnspecified Functional Bowel DisorderRome IV — bowel symptoms fitting no other categoryCentrally Mediated Abdominal Pain (CAPS)Rome IV — continuous pain unrelated to gut eventsNarcotic Bowel SyndromeRome IV — opioid-induced hyperalgesia of the gutFaecal Incontinence (Rome IV)Rome IV — the criteria, and why nobody is askedFunctional Anorectal PainLevator ani, unspecified pain and proctalgia fugaxFunctional Defecation DisordersRome IV — dyssynergia and inadequate propulsionInfant RegurgitationRome IV — the happy spitter, and the alarm features that rule it outInfant ColicRome IV — recurrent unexplained crying in a well infant under 5 monthsInfant DyscheziaRome IV — straining before a soft stool, and why not to intervenePaediatric Functional ConstipationRome IV — two of six over one month, with overflow soiling as a criterionToddler's DiarrhoeaRome IV functional diarrhoea of childhood — painless, thriving childPaediatric Cyclic Vomiting SyndromeRome IV — both age bands, with different criteria for eachPaediatric Rumination SyndromeRome IV — infant and child/adolescent criteriaFunctional Nausea & Vomiting (Children)Rome IV — two separate disorders that can be met togetherAerophagiaRome IV — distension that increases through the dayPaediatric Functional DyspepsiaRome IV — four times a month, with PDS and EPS subtypingPaediatric Irritable Bowel SyndromeRome IV — plus the constipation clause clinicians missAbdominal MigraineRome IV — stereotypical incapacitating episodes weeks apartFunctional Abdominal Pain — NOSRome IV — the residual category, reached after the other threeNonretentive Faecal IncontinenceRome IV — soiling without retention, where laxatives make it worseFunctional DyspepsiaRome IV — with PDS and EPS subtypingRumination SyndromeRome IV — effortless regurgitation without retchingCyclic Vomiting SyndromeRome IV — stereotypical episodic vomitingCannabinoid HyperemesisRome IV — CVS pattern relieved by cannabis cessationChronic Nausea & VomitingRome IV — chronic nausea and vomiting syndromeBelching DisordersRome IV — supragastric vs gastric belchingFunctional HeartburnRome IV — heartburn with normal acid exposureReflux HypersensitivityRome IV — normal acid exposure, positive symptom associationFunctional Chest PainRome IV — non-cardiac, non-reflux chest painGlobusRome IV — painless lump-in-throat sensationFunctional DysphagiaRome IV — dysphagia with normal endoscopy and manometry
  1. Calculators
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  3. Globus
Functional GI

Globus

Rome IV — painless lump-in-throat sensation

Rome IV applies the same duration rule to every functional oesophageal disorder. Onset within the last six months does not meet criteria however typical the symptoms are.

Between meals is diagnostic. A sensation that appears only while swallowing is dysphagia, not globus.

Either symptom mandates investigation for a structural or mucosal cause and excludes globus.

Added in Rome IV. Heterotopic gastric mucosa at the upper oesophageal sphincter can generate globus and is treatable endoscopically, so it has to be looked for specifically on withdrawal.

A painless lump in the throat, felt between meals and not on swallowing. Any true dysphagia or odynophagia takes the patient out of this diagnosis and into investigation.

When to use
Use it in a patient describing a lump, tightness or something stuck in the throat that is not painful and does not interfere with swallowing, once endoscopy has been done. The criteria are most valuable for what they force you to check: the distinction between globus and true dysphagia, which the patient will not reliably make for you, and the presence of a gastric inlet patch, which requires deliberate inspection of the proximal oesophagus on withdrawal. It does not apply to a patient with any difficulty swallowing, any pain on swallowing, or any weight loss — all of which take the presentation out of globus and into investigation for a structural cause.
Why use it
Because globus is common, benign and frightening, and the default management is often a proton pump inhibitor prescribed indefinitely without objective evidence of reflux and without benefit. The criteria redirect the assessment toward the two things that actually change management: excluding true dysphagia, which is a red flag and mandates investigation, and looking specifically for a gastric inlet patch, which is treatable endoscopically and which Rome IV added precisely because it is a recognised and reversible cause of the symptom. Beyond that, a positive diagnosis matters here more than in most functional disorders, because the fear that drives repeat presentation in globus is fear of cancer, and reassurance only works when it follows an explanation the patient can accept.
Formula, evidence and interpretation

About the Rome IV Criteria for Globus

Between meals is the diagnostic detail. Globus is a persistent or intermittent painless sensation of a lump or foreign body in the throat that occurs *between* meals rather than during swallowing — a sensation felt while swallowing is dysphagia and a different problem entirely. Rome IV requires that sensation at least once a week over three months with onset at least six months ago, together with the absence of dysphagia and odynophagia, no gastric inlet patch in the proximal oesophagus, no evidence that reflux disease or eosinophilic oesophagitis is responsible, and no major oesophageal motor disorder. The inlet patch exclusion is new in Rome IV and is the one most often overlooked.

On this page

  • Formula
  • Interpreting the result
  • Inputs
  • What it returns
  • How it is calculated
  • Facts & figures
  • Evidence
  • How it compares
  • Pearls & pitfalls
  • Critical actions
  • Why it exists
  • About the creator
  • Limitations
  • If you are the patient
  • FAQ
  • Related calculators
  • References

Formula

Globus = ALL of: painless lump or foreign-body sensation, occurring between meals AND absence of dysphagia and odynophagia AND no gastric inlet patch in the proximal oesophagus AND no evidence that reflux disease or eosinophilic oesophagitis is the cause AND no major oesophageal motor disorder AND criteria fulfilled for the last 3 months, onset >= 6 months ago, at least once a week
Between meals
The discriminating phrase. A sensation occurring during swallowing is dysphagia; globus is felt when not swallowing.
Painless
Pain on swallowing is odynophagia and is an explicit exclusion.
Gastric inlet patch
Heterotopic gastric mucosa in the proximal oesophagus, newly excluded in Rome IV after evidence linked it to globus symptom generation.
  • Between meals is the discriminating word — a sensation occurring during swallowing is dysphagia, not globus.
  • The gastric inlet patch exclusion is new in Rome IV, added after evidence linked heterotopic gastric mucosa to globus symptoms.

Interpreting the result

Meeting the criteria supports a positive, benign diagnosis and the work then shifts to explanation. Globus responds better to a clear account and planned follow-up than to medication, and the explanation that lands is usually about heightened awareness of normal throat sensation combined with muscle tension, reinforced by the checking behaviours — repeated swallowing, throat clearing — that the sensation provokes and that in turn perpetuate it. Speech and language therapy has evidence here and is substantially under-used. Empirical proton pump inhibitors are widely prescribed and perform poorly without objective reflux; if a trial is given, it should be time-limited and stopped on non-response rather than continued indefinitely. Where criteria are not met, the reason matters enormously: coexisting dysphagia or odynophagia is a red flag requiring investigation, while an unexamined proximal oesophagus is simply an incomplete endoscopy that should be repeated with the inlet patch specifically in mind.

ScoreBandWhat it meansAction
All criteria metGlobusA benign, painless throat sensation between meals with structural, mucosal, motor and inlet-patch causes excludedExplain and reassure with planned follow-up; consider speech and language therapy. Avoid open-ended acid suppression
Dysphagia or odynophagia presentCriteria not met — red flagDifficulty or pain on swallowing takes the presentation out of globus entirelyInvestigate for a structural or mucosal cause; do not reassure on the basis of a globus label
Other criterion unmetCriteria not metUsually an incomplete endoscopy — most often the proximal oesophagus not inspected for an inlet patchComplete the outstanding assessment before settling on or excluding the diagnosis

What the Globus needs (6 inputs)

Timing and frequency
Criteria fulfilled for the last three months, onset at least six months before diagnosis, occurring at least once a week.
Painless lump or foreign-body sensation, occurring between meals
Both qualifiers matter. Painless separates it from odynophagia, and between meals separates it from dysphagia — globus characteristically eases or disappears while actually eating and drinking, which patients often find counter-intuitive enough not to mention unless asked.
Absence of dysphagia and odynophagia
Either symptom excludes globus and mandates investigation for a structural or mucosal cause. This is the criterion that carries the safety of the diagnosis.
No gastric inlet patch in the proximal oesophagus
Heterotopic gastric mucosa just below the upper oesophageal sphincter, which can secrete acid locally and generate globus. Added as an explicit exclusion in Rome IV, and easily missed because it requires deliberate inspection during withdrawal rather than appearing on a standard survey.
No evidence that reflux disease or eosinophilic oesophagitis is the cause
Endoscopy with oesophageal biopsies. Reflux is frequently blamed for globus without objective evidence, which is why the criterion asks for evidence rather than plausibility.
No major oesophageal motor disorder
Achalasia, EGJ outflow obstruction, distal oesophageal spasm, jackhammer oesophagus or absent contractility on high-resolution manometry.

What it returns

Criteria met or not met
All six requirements including the timing rule must hold simultaneously.
Which criteria remain outstanding
Named explicitly. Coexisting dysphagia and an unexamined proximal oesophagus are the two most common failures, and they have very different implications.

How it is calculated

The criteria are built around distinguishing a benign sensation from a dangerous one, and the sorting is done by the accompanying features rather than by the sensation itself. A lump in the throat with normal swallowing is globus; the same lump with impaired swallowing is dysphagia and demands a different pathway. Rome IV then layers on exclusions for the conditions that can generate a genuine globus sensation and are treatable — reflux disease, eosinophilic oesophagitis, major motor disorders, and, newly, the gastric inlet patch. The addition of the inlet patch is the substantive change from Rome III and reflects accumulating evidence that heterotopic gastric mucosa at the upper oesophageal sphincter can secrete acid locally, produce exactly this symptom, and respond to endoscopic ablation. Including it turned an exclusively functional label into one with a treatable differential that has to be actively excluded.

Facts & figures

Globus or dysphagia — the distinction the criteria turn on
FeatureGlobusDysphagia
When it is feltBetween mealsDuring swallowing
Effect of eating and drinkingOften eases or disappearsProvokes or worsens it
PainAbsent by definitionMay be present (odynophagia)
Weight lossNot expectedA red flag when present
ImplicationBenign; explanation and follow-upInvestigate for structural or mucosal cause

Patients frequently describe both as 'something stuck', so the history has to ask specifically whether the sensation is present between meals and whether food actually holds up. The two are not variations of one complaint.

The gastric inlet patch — why Rome IV added it
AspectDetail
What it isHeterotopic gastric mucosa in the proximal oesophagus, just below the upper oesophageal sphincter
Why it matters hereCan secrete acid locally and generate a globus sensation, sometimes with throat clearing and cough
Why it is missedRequires deliberate inspection on withdrawal; it sits above the level most endoscopists survey carefully
Why it changes managementTreatable endoscopically, in contrast to functional globus

This is the substantive change from Rome III. A globus diagnosis made without the proximal oesophagus having been examined has skipped the one exclusion with a specific treatment attached.

Evidence

Derivation — Rome Foundation, oesophageal disorders committee

2016

Consensus criteria produced by the Rome IV oesophageal disorders committee and published in Gastroenterology in 2016, revising Rome III.

Consensus-derived with no discrimination statistics. The substantive revision from Rome III was the explicit exclusion of the gastric inlet patch, added in response to evidence linking heterotopic gastric mucosa to globus symptom generation.

Reflux exclusion framework — Lyon Consensus 2.0

2024

International consensus defining conclusive evidence for and against gastro-oesophageal reflux disease.

Provides the thresholds by which reflux can be excluded as the cause, which matters in globus because reflux is so frequently assumed rather than demonstrated.

Motor disorder framework — Chicago Classification v4.0

2021

International consensus on oesophageal motility disorders defined by high-resolution manometry.

Defines the major motor disorders that must be absent, including EGJ outflow obstruction and achalasia, which can present with an upper-throat sensation as well as with dysphagia.

How it compares

Globus vs Functional dysphagia

Mutually exclusive by definition — globus requires the absence of dysphagia, so a patient with food actually sticking is assessed as functional dysphagia instead.

The symptoms sound similar when patients describe them and are entirely different clinically. Globus is felt between meals and often eases while eating; functional dysphagia is the sense of food lodging or passing abnormally during swallowing. The investigation burden differs accordingly: functional dysphagia demands high-resolution manometry to exclude achalasia, whereas globus centres on endoscopy with careful inspection of the proximal oesophagus. Getting the history distinction wrong routes the patient down the wrong pathway, and in the direction of under-investigating dysphagia it is the more dangerous error.

Open the Functional dysphagia calculator →

Globus vs Gastric inlet patch

Not a competing diagnosis but a required exclusion — and the only cause of a globus sensation with a specific endoscopic treatment.

Heterotopic gastric mucosa in the proximal oesophagus can secrete acid locally and produce globus, throat clearing and cough. Rome IV made its absence an explicit criterion because it is both a genuine cause and a routinely missed one: it sits just below the upper oesophageal sphincter, an area passed over quickly on intubation and often not re-examined on withdrawal. Where it is found and symptomatic, endoscopic ablation is an option that functional globus has no equivalent to. A globus diagnosis recorded without the proximal oesophagus having been looked at has skipped the exclusion that could have been treated.

Globus vs Laryngopharyngeal reflux

An overlapping and contested label — Rome IV requires evidence that reflux is the cause rather than accepting the assumption that underpins most LPR diagnoses.

Laryngopharyngeal reflux is frequently diagnosed on symptoms plus laryngoscopic signs whose specificity is poor, and it is the usual justification for the long-term proton pump inhibitor many globus patients end up taking. The Rome IV criterion is deliberately stricter: reflux must be shown to be the cause, not merely plausible. In practice, patients labelled with laryngopharyngeal reflux who have no objective reflux on monitoring and no response to acid suppression are better served by the globus pathway — explanation, speech and language therapy, and stopping the ineffective drug — than by escalating treatment for a diagnosis that has not been demonstrated.

Pearls & pitfalls

  • Between meals is the diagnostic feature. A sensation felt only while swallowing is dysphagia, and the two are not variations of the same complaint.
  • Globus characteristically eases while eating and drinking. Patients rarely volunteer this because it seems paradoxical to them — ask directly.
  • Any dysphagia or odynophagia excludes globus and mandates investigation. This is the criterion that keeps the diagnosis safe.
  • Look for a gastric inlet patch specifically, on withdrawal. It sits just below the upper oesophageal sphincter, above where most examinations are careful, and it is treatable — which is why Rome IV added it.
  • Reflux is blamed for globus far more often than it is demonstrated. The criterion requires evidence, not plausibility.
  • Biopsies are required to exclude eosinophilic oesophagitis, which cannot be ruled out on appearance.
  • Empirical proton pump inhibitors perform poorly in globus without objective reflux. If tried, set a stop date rather than continuing indefinitely on no response.
  • Speech and language therapy has evidence in globus and is markedly under-used compared with acid suppression.
  • The old name globus hystericus is both inaccurate and harmful. Anxiety commonly accompanies globus and worsens it, but the sensation is not manufactured by it.
  • Weight loss, progressive symptoms or a neck mass are not part of globus at any frequency and require investigation regardless of how well the other criteria fit.

Critical actions

  • Establish explicitly whether the sensation occurs between meals and whether food actually holds up — that history distinguishes globus from dysphagia and cannot be inferred.
  • Perform endoscopy, inspecting the proximal oesophagus deliberately on withdrawal for a gastric inlet patch.
  • Take oesophageal biopsies to exclude eosinophilic oesophagitis.
  • Examine the neck and thyroid, and consider ENT assessment where the sensation is localised high or there is any voice change.
  • Investigate rather than reassure if dysphagia, odynophagia, weight loss or progression is present.
  • Give a positive explanation covering heightened throat awareness, muscle tension, and the way repeated swallowing and throat clearing perpetuate the sensation.
  • Refer for speech and language therapy where symptoms persist, rather than defaulting to longer acid suppression.
  • If a proton pump inhibitor trial is given, time-limit it and stop it on non-response.

Why this score exists

The most interesting thing about the Rome IV globus criteria is what the committee chose to add rather than what it kept. Globus has one of the longest and least helpful histories in medicine — it was globus hystericus for most of the twentieth century, a name that told the patient the problem was in their mind and told the clinician not to look further. The addition of the gastric inlet patch exclusion moves in exactly the opposite direction: it says there is a specific, endoscopically visible, endoscopically treatable cause of this symptom that has been routinely missed, and it must be excluded before the functional label is applied. The committee also pushed for better endoscopic evaluation of the oropharynx generally. Taken together the revision reframes globus from a diagnosis of psychological attribution to one that requires a careful look at a part of the oesophagus most examinations pass over.

About the creator

  • Qasim Aziz

    First author, Rome IV oesophageal disorders committee

    Chaired the committee that produced the Rome IV functional oesophageal disorder criteria.

  • Hiroto Miwa

    Co-author, Rome IV oesophageal disorders committee

    Co-authored the Rome IV oesophageal chapter, including the revised globus criteria.

  • Frank Zerbib

    Co-author; oesophageal physiology

    Co-authored the chapter and contributed to the physiological assessment underpinning the exclusions.

Limitations

  • Relies on a history distinction — between meals versus during swallowing — that patients often cannot make unprompted, so the criteria are only as good as the questions asked.
  • The gastric inlet patch exclusion requires deliberate proximal inspection that many endoscopies do not include, so it is frequently recorded as absent without having been looked for.
  • Requires endoscopy and, strictly, manometry, which is often not performed in practice for a symptom regarded as benign.
  • Agreed by committee, with no discrimination statistics; the diagnosis rests entirely on what a normal examination and normal manometry fail to explain.
  • Does not address the anxiety and cancer fear that accompany globus in a large proportion of patients and drive much of the repeat presentation.
  • Silent on ENT and laryngeal causes, which sit outside the gastroenterological scope but overlap substantially with this presentation.
  • Says nothing about severity — the sensation ranges from a background awareness to a constant preoccupation, and the criteria treat those identically.
  • The evidence base for treatment in globus specifically, as opposed to functional oesophageal disorders generally, remains thin.

If you are the patient

Globus is the feeling of a lump, tightness or something stuck in the throat when there is nothing there. The key feature is that it is not painful and it does not stop you swallowing — in fact, most people find it eases while they are actually eating or drinking and comes back afterwards, which surprises them. It is common and it is benign, but it is also genuinely unpleasant, and the worry that it might be cancer is what brings most people to a doctor. Your doctor will check for other causes, including a camera test that should include a careful look at the top of the gullet for a small patch of stomach-type lining that can occasionally sit there and cause exactly this feeling — that one is treatable. If everything is clear, the explanation is that the throat has become over-aware of normal sensation, often with some muscle tension, and that the natural response of swallowing repeatedly or clearing the throat keeps the feeling going. Speech and language therapy genuinely helps and is worth asking about; it is offered far less often than it should be. Acid-reducing tablets are commonly prescribed but work poorly unless reflux has actually been shown, so if you have been on them for months without benefit it is reasonable to ask whether they should continue. Do go back promptly if you develop difficulty swallowing, pain on swallowing, weight loss, or a lump in the neck — those are different symptoms and need looking into.

Frequently asked questions

What are the Rome IV criteria for globus?#

A persistent or intermittent painless sensation of a lump or foreign body in the throat, occurring between meals, with no dysphagia or odynophagia; no gastric inlet patch in the proximal oesophagus; no evidence that reflux disease or eosinophilic oesophagitis is the cause; and no major oesophageal motor disorder. Criteria must be met for three months with onset at least six months earlier, at least once a week.

What is the difference between globus and dysphagia?#

Timing. Globus is felt between meals and typically eases while eating or drinking; dysphagia is the sensation of food actually sticking during swallowing. Globus is painless and benign, while dysphagia is a red flag that requires investigation for a structural or mucosal cause. Patients often describe both as 'something stuck', so the history has to ask specifically.

Why does Rome IV exclude a gastric inlet patch in globus?#

Because heterotopic gastric mucosa sitting just below the upper oesophageal sphincter can secrete acid locally and generate exactly this sensation — and unlike functional globus, it can be treated endoscopically. It was added in Rome IV on the strength of evidence linking it to globus symptom generation. It is easily missed because it requires deliberate inspection on withdrawal rather than appearing on a standard survey.

Do proton pump inhibitors help globus?#

Poorly, unless objective reflux has been demonstrated. They are very commonly prescribed empirically and very commonly continued indefinitely without benefit. Rome IV requires evidence that reflux is the cause rather than the assumption that it might be. If a trial is given it should be time-limited and stopped on non-response.

Is globus caused by anxiety?#

Anxiety commonly accompanies globus and worsens it, but it does not manufacture the sensation. The old name 'globus hystericus' was both inaccurate and harmful, because it told patients the symptom was imagined and told clinicians not to look further. The current understanding is heightened awareness of normal throat sensation with muscle tension, perpetuated by repeated swallowing and throat clearing.

What treatment works for globus?#

Explanation with planned follow-up does more than any drug, because the fear of cancer is what drives most of the distress. Speech and language therapy has evidence and is substantially under-used. Neuromodulators can be tried where symptoms are persistent and troublesome. Long-term acid suppression without demonstrated reflux is not supported.

When should a lump-in-throat sensation be investigated urgently?#

When there is difficulty swallowing, pain on swallowing, weight loss, progressive symptoms, a neck mass or voice change. None of those is part of globus at any frequency, and their presence excludes the diagnosis under Rome IV rather than qualifying it. A globus label should never be used to justify not investigating these.

How often does the sensation need to occur to meet the criteria?#

At least once a week over the last three months, with onset at least six months before diagnosis — the same threshold Rome IV applies to functional chest pain and functional dysphagia, and lower than the twice-weekly requirement for functional heartburn and reflux hypersensitivity.

Related calculators

  • Functional Dysphagia — Rome IV — dysphagia with normal endoscopy and manometry
  • Functional Heartburn — Rome IV — heartburn with normal acid exposure
  • Reflux Hypersensitivity — Rome IV — normal acid exposure, positive symptom association
  • Functional Chest Pain — Rome IV — non-cardiac, non-reflux chest pain
  • Rome IV Criteria for IBS — Irritable bowel syndrome diagnosis and subtype

References

Original / primary reference

  1. Aziz Q, Fass R, Gyawali CP, Miwa H, Pandolfino JE, Zerbib F. Esophageal Disorders. Gastroenterology. 2016;150(6):1368-1379 (Rome IV).

Exclusion frameworks

  1. Gyawali CP, Yadlapati R, Fass R, et al. Updates to the modern diagnosis of GERD: Lyon consensus 2.0. Gut. 2024;73(2):361-371.
  2. Yadlapati R, Kahrilas PJ, Fox MR, et al. Esophageal motility disorders on high-resolution manometry: Chicago classification version 4.0. Neurogastroenterol Motil. 2021;33(1):e14058.

Last updated July 31, 2026. Clinical knowledge base written and curated by GastroAGI Team from primary medical literature.

Written from primary literature and not yet independently clinically reviewed.

For use by qualified healthcare professionals. This calculator supports clinical judgement and does not replace it.